Lisha Qi

Summary

Country: China

Publications

  1. pmc Wnt3a expression is associated with epithelial-mesenchymal transition and promotes colon cancer progression
    Lisha Qi
    Department of Pathology, Tianjin Medical University Cancer Institute and Hospital, Tianjin, 300060, China
    J Exp Clin Cancer Res 33:107. 2014
  2. pmc Wnt3a Promotes the Vasculogenic Mimicry Formation of Colon Cancer via Wnt/β-Catenin Signaling
    Lisha Qi
    Department of Pathology, Tianjin Medical University Cancer Institute and Hospital, Tianjin 300060, China
    Int J Mol Sci 16:18564-79. 2015
  3. doi request reprint Dickkopf-1 inhibits epithelial-mesenchymal transition of colon cancer cells and contributes to colon cancer suppression
    Lisha Qi
    Department of Pathology, Tianjin Medical University Cancer Institute and Hospital, Tianjin, China
    Cancer Sci 103:828-35. 2012
  4. doi request reprint Wnt5a suppresses colon cancer by inhibiting cell proliferation and epithelial-mesenchymal transition
    Runfen Cheng
    Department of Pathology, Tianjin Medical University Cancer Institute and Hospital, Tianjin, 300060, China
    J Cell Physiol 229:1908-17. 2014
  5. doi request reprint Dickkopf-1 expression is down-regulated during the colorectal adenoma-carcinoma sequence and correlates with reduced microvessel density and VEGF expression
    Zhiyong Liu
    Department of Pathology, Tianjin Medical University Cancer Institute and Hospital, Tianjin, China
    Histopathology 67:158-66. 2015
  6. pmc Transforming growth factor-β1 induces EMT by the transactivation of epidermal growth factor signaling through HA/CD44 in lung and breast cancer cells
    Lingmei Li
    Department of Pathology, Tianjin Medical University Cancer Institute and Hospital, Tianjin 300060, P R China
    Int J Mol Med 36:113-22. 2015
  7. doi request reprint Zinc finger E-box binding homeobox 1 promotes vasculogenic mimicry in colorectal cancer through induction of epithelial-to-mesenchymal transition
    Zhiyong Liu
    Department of Pathology, Tianjin Cancer Institute and Hospital, Tianjin Medical University, Tianjin, China
    Cancer Sci 103:813-20. 2012
  8. pmc Targeting EZH2 regulates tumor growth and apoptosis through modulating mitochondria dependent cell-death pathway in HNSCC
    Xuan Zhou
    Department of Maxillofacial and Otorhinolaryngology Oncology, Tianjin Medical University Cancer Institute and Hospital, Key Laboratory of Cancer Prevention and Therapy, Tianjin Cancer Institute, National Clinical Research Center of Cancer, Tianjin, China
    Oncotarget 6:33720-32. 2015

Collaborators

  • Yu Wang
  • Bin Zhang
  • Xiulan Zhao
  • Jun Gao
  • Min Li
  • Zhiyong Liu
  • Baocun Sun
  • Lingmei Li
  • Xuan Zhou
  • Wangzhao Song
  • Yixian Li
  • Runfen Cheng
  • Guoshuai Cai
  • Rui Jin
  • Yanhui Zhang
  • Xudong Wang
  • Yanxue Liu
  • Shanshan Sun
  • Lun Zhang
  • Lingping Kong
  • Danfang Zhang
  • Chunsheng Kang
  • Mei Mei
  • Wenfeng Cao
  • Yu Ren
  • Zhijie Liang
  • Qiang Gu
  • Hui Li
  • Xue Leng

Detail Information

Publications8

  1. pmc Wnt3a expression is associated with epithelial-mesenchymal transition and promotes colon cancer progression
    Lisha Qi
    Department of Pathology, Tianjin Medical University Cancer Institute and Hospital, Tianjin, 300060, China
    J Exp Clin Cancer Res 33:107. 2014
    ..However, the roles of Wnt3a, the representative canonical Wnt ligand, in EMT and colon cancer progression have not yet been fully explored...
  2. pmc Wnt3a Promotes the Vasculogenic Mimicry Formation of Colon Cancer via Wnt/β-Catenin Signaling
    Lisha Qi
    Department of Pathology, Tianjin Medical University Cancer Institute and Hospital, Tianjin 300060, China
    Int J Mol Sci 16:18564-79. 2015
    ..Taken together, our results suggest that Wnt/β-catenin signaling is involved in VM formation in colon cancer and might contribute to the development of more accurate treatment modalities aimed at VM...
  3. doi request reprint Dickkopf-1 inhibits epithelial-mesenchymal transition of colon cancer cells and contributes to colon cancer suppression
    Lisha Qi
    Department of Pathology, Tianjin Medical University Cancer Institute and Hospital, Tianjin, China
    Cancer Sci 103:828-35. 2012
    ..Our findings indicate that Dkk1 can suppress the progression of colon cancer, possibly through EMT inhibition, and could therefore serve as a target for tumor therapy...
  4. doi request reprint Wnt5a suppresses colon cancer by inhibiting cell proliferation and epithelial-mesenchymal transition
    Runfen Cheng
    Department of Pathology, Tianjin Medical University Cancer Institute and Hospital, Tianjin, 300060, China
    J Cell Physiol 229:1908-17. 2014
    ..Wnt5a could be used as a novel prognostic marker and/or therapeutic target for colon cancer in the future...
  5. doi request reprint Dickkopf-1 expression is down-regulated during the colorectal adenoma-carcinoma sequence and correlates with reduced microvessel density and VEGF expression
    Zhiyong Liu
    Department of Pathology, Tianjin Medical University Cancer Institute and Hospital, Tianjin, China
    Histopathology 67:158-66. 2015
    ..However, little is known about the role of Dkk1 during the colorectal adenoma-carcinoma sequence. This study aimed to elucidate the role of Dkk1 in tumorigenesis and angiogenesis in colorectal cancer...
  6. pmc Transforming growth factor-β1 induces EMT by the transactivation of epidermal growth factor signaling through HA/CD44 in lung and breast cancer cells
    Lingmei Li
    Department of Pathology, Tianjin Medical University Cancer Institute and Hospital, Tianjin 300060, P R China
    Int J Mol Med 36:113-22. 2015
    ..In conclusion, our data demonstrate that TGF-β1 induces EMT by the transactivation of EGF signaling through HA/CD44 in lung and breast cancer cells...
  7. doi request reprint Zinc finger E-box binding homeobox 1 promotes vasculogenic mimicry in colorectal cancer through induction of epithelial-to-mesenchymal transition
    Zhiyong Liu
    Department of Pathology, Tianjin Cancer Institute and Hospital, Tianjin Medical University, Tianjin, China
    Cancer Sci 103:813-20. 2012
    ..Taken together, our results suggest that ZEB1 can promote VM formation by inducing EMT in CRC and might represent an important target in CRC...
  8. pmc Targeting EZH2 regulates tumor growth and apoptosis through modulating mitochondria dependent cell-death pathway in HNSCC
    Xuan Zhou
    Department of Maxillofacial and Otorhinolaryngology Oncology, Tianjin Medical University Cancer Institute and Hospital, Key Laboratory of Cancer Prevention and Therapy, Tianjin Cancer Institute, National Clinical Research Center of Cancer, Tianjin, China
    Oncotarget 6:33720-32. 2015
    ..A xenograft tumor model was used to confirm that EZH2 depletion inhibited HNSCC cell growth and induced tumor cell apoptosis. In summary, EZH2 is a potential anti-tumor target in HNSCC. ..