Michael E Feigin

Summary

Affiliation: State University of New York
Country: USA

Publications

  1. ncbi RGS19 regulates Wnt-beta-catenin signaling through inactivation of Galpha(o)
    Michael E Feigin
    Department of Pharmacology, Health Sciences Center, State University of New York at Stony Brook, Stony Brook, NY 11794 8651, USA
    J Cell Sci 120:3404-14. 2007
  2. doi G alpha o mediates WNT-JNK signaling through dishevelled 1 and 3, RhoA family members, and MEKK 1 and 4 in mammalian cells
    Rama Kamesh Bikkavilli
    Department of Pharmacology, Health Sciences Center, State University of New York at Stony Brook, Stony Brook, NY 11794 8651, USA
    J Cell Sci 121:234-45. 2008
  3. doi p38 mitogen-activated protein kinase regulates canonical Wnt-beta-catenin signaling by inactivation of GSK3beta
    Rama Kamesh Bikkavilli
    Department of Pharmacology, Health Sciences Center, State University of New York at Stony Brook, Stony Brook, NY 11794 8651, USA
    J Cell Sci 121:3598-607. 2008
  4. doi OSTM1 regulates beta-catenin/Lef1 interaction and is required for Wnt/beta-catenin signaling
    Michael E Feigin
    Cold Spring Harbor Laboratory, One Bungtown Road, Cold Spring Harbor, NY 11724, United States
    Cell Signal 20:949-57. 2008

Collaborators

Detail Information

Publications4

  1. ncbi RGS19 regulates Wnt-beta-catenin signaling through inactivation of Galpha(o)
    Michael E Feigin
    Department of Pharmacology, Health Sciences Center, State University of New York at Stony Brook, Stony Brook, NY 11794 8651, USA
    J Cell Sci 120:3404-14. 2007
    ..However, knockdown of RGS19 by siRNA suppressed canonical Wnt signaling, suggesting a complex role for RGS19 in regulating the ability of Wnt3a to signal to the level of beta-catenin and gene transcription...
  2. doi G alpha o mediates WNT-JNK signaling through dishevelled 1 and 3, RhoA family members, and MEKK 1 and 4 in mammalian cells
    Rama Kamesh Bikkavilli
    Department of Pharmacology, Health Sciences Center, State University of New York at Stony Brook, Stony Brook, NY 11794 8651, USA
    J Cell Sci 121:234-45. 2008
    ..These data reveal both common and unique signaling elements in WNT3a-sensitive pathways, highlighting crosstalk from WNT3a-JNK to WNT3a-beta-catenin signaling...
  3. doi p38 mitogen-activated protein kinase regulates canonical Wnt-beta-catenin signaling by inactivation of GSK3beta
    Rama Kamesh Bikkavilli
    Department of Pharmacology, Health Sciences Center, State University of New York at Stony Brook, Stony Brook, NY 11794 8651, USA
    J Cell Sci 121:3598-607. 2008
    ..We demonstrate the involvement of G-proteins and Dishevelleds in Wnt3a-induced p38 MAPK activation, highlighting a critical role for p38 MAPK in canonical Wnt-beta-catenin signaling...
  4. doi OSTM1 regulates beta-catenin/Lef1 interaction and is required for Wnt/beta-catenin signaling
    Michael E Feigin
    Cold Spring Harbor Laboratory, One Bungtown Road, Cold Spring Harbor, NY 11724, United States
    Cell Signal 20:949-57. 2008
    ....