Claudia Wietek

Summary

Publications

  1. ncbi request reprint IkappaB kinase epsilon interacts with p52 and promotes transactivation via p65
    Claudia Wietek
    School of Biochemistry and Immunology, Trinity College, Dublin 2, Ireland
    J Biol Chem 281:34973-81. 2006
  2. ncbi request reprint Diversity and regulation in the NF-kappaB system
    Claudia Wietek
    School of Biochemistry and Immunology, Trinity College Dublin, Dublin 2, Ireland
    Trends Biochem Sci 32:311-9. 2007
  3. ncbi request reprint Interferon regulatory factor-3-mediated activation of the interferon-sensitive response element by Toll-like receptor (TLR) 4 but not TLR3 requires the p65 subunit of NF-kappa
    Claudia Wietek
    Department of Biochemistry, Trinity College, Dublin, Ireland
    J Biol Chem 278:50923-31. 2003

Detail Information

Publications3

  1. ncbi request reprint IkappaB kinase epsilon interacts with p52 and promotes transactivation via p65
    Claudia Wietek
    School of Biochemistry and Immunology, Trinity College, Dublin 2, Ireland
    J Biol Chem 281:34973-81. 2006
    ..These novel interactions reveal a hitherto unknown function of IKKepsilon in the regulation of the alternative NF-kappaB activation pathway involving p52 and p65...
  2. ncbi request reprint Diversity and regulation in the NF-kappaB system
    Claudia Wietek
    School of Biochemistry and Immunology, Trinity College Dublin, Dublin 2, Ireland
    Trends Biochem Sci 32:311-9. 2007
    ..Furthermore, the state of a cell and its cellular background in addition to overall promoter structure and variations in the kappaB target sequence will all define the composition and activity of multimeric NF-kappaB complexes...
  3. ncbi request reprint Interferon regulatory factor-3-mediated activation of the interferon-sensitive response element by Toll-like receptor (TLR) 4 but not TLR3 requires the p65 subunit of NF-kappa
    Claudia Wietek
    Department of Biochemistry, Trinity College, Dublin, Ireland
    J Biol Chem 278:50923-31. 2003
    ..Finally, p65 promoted transactivation of gene expression by IRF-3. These results therefore indicate that IRF-3-mediated activation of the ISRE by TLR4 but not TLR3 requires the p65 subunit of NF-kappa B...